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Ion channels boost axonal injury in multiple sclerosis

Nat Med. 18(12):1743-5 (2012)

Authors/Editors: Hohlfeld R.
Publication Date: 2012

HOHLFELD-nature-medicine-2012

Abstract

In neuroinflammatory diseases such as multiple sclerosis, ion channels may fan the embers of neurodegeneration.
A new study shows that the cation channel TRPM4 (transient receptor potential melastatin 4) crucially contributes to axonal loss in an animal model of multiple sclerosis.

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